Predicted to enable O-acyltransferase activity and lysophospholipid acyltransferase activity. Predicted to be involved in glycerophospholipid metabolic process and regulation of triglyceride metabolic process. Predicted to act upstream of or within layer formation in cerebral cortex; phosphatidylinositol metabolic process; and ventricular system development. Predicted to be located in endoplasmic reticulum; membrane; and mitochondria-associated endoplasmic reticulum membrane contact site. Human ortholog(s) of this gene implicated in autosomal recessive intellectual developmental disorder 57. Orthologous to human MBOAT7 (membrane bound O-acyltransferase domain containing 7); INTERACTS WITH 3H-1,2-dithiole-3-thione; 4,4'-sulfonyldiphenol; amphetamine.
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol F] results in increased expression of MBOAT7 mRNA
[bisphenol A co-treated with bisphenol F co-treated with bisphenol S] results in increased expression of MBOAT7 mRNA and [bisphenol A co-treated with Genistein] results in decreased methylation of MBOAT7 gene
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol F] results in increased expression of MBOAT7 mRNA
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol F] results in increased expression of MBOAT7 mRNA
[Methionine deficiency co-treated with Choline deficiency co-treated with Folic Acid deficiency] results in decreased methylation of MBOAT7 gene more ...
[INS protein co-treated with Dexamethasone co-treated with 1-Methyl-3-isobutylxanthine co-treated with Indomethacin co-treated with bisphenol F] results in increased expression of MBOAT7 mRNA
