GENE - CHEMICAL INTERACTIONS REPORT
Note: Use of the qualifier "multiple interactions" designates that the annotated interaction
is comprised of a complex set of reactions and/or regulatory events, possibly involving
additional chemicals and/or gene products.
Object Symbol | Qualifier | Evidence | With | Reference | Source | Notes | Original Reference(s) | Atg5 | multiple interactions | EXP | | 6480464 | CTD | [TRP53BP2 protein affects the susceptibility to Carbon Tetrachloride] which affects the expression of ATG5 protein; ATG5 protein inhibits the reaction [curcumol inhibits the reaction [Carbon Tetrachloride results in increased activity of GPT protein]]; ATG5 protein inhibits the reaction [curcumol inhibits the reaction [Carbon Tetrachloride results in increased expression of CD34 protein]]; ATG5 protein inhibits the reaction [curcumol inhibits the reaction [Carbon Tetrachloride results in increased expression of KDR protein]]; ATG5 protein inhibits the reaction [curcumol inhibits the reaction [Carbon Tetrachloride results in increased expression of PECAM1 protein]]; ATG5 protein inhibits the reaction [curcumol inhibits the reaction [Carbon Tetrachloride results in increased expression of VEGFA protein]]; ATG5 protein inhibits the reaction [curcumol inhibits the reaction [Carbon Tetrachloride results in increased expression of VWF protein]]; ATG5 protein promotes the reaction [Carbon Tetrachloride results in increased activity of GPT protein]; ATG5 protein promotes the reaction [Carbon Tetrachloride results in increased expression of CD34 protein]; ATG5 protein promotes the reaction [Carbon Tetrachloride results in increased expression of KDR protein]; ATG5 protein promotes the reaction [Carbon Tetrachloride results in increased expression of PECAM1 protein]; ATG5 protein promotes the reaction [Carbon Tetrachloride results in increased expression of VEGFA protein]; ATG5 protein promotes the reaction [Carbon Tetrachloride results in increased expression of VWF protein] | PMID:31513885 PMID:33549628 | | ||||||||||||||||||||||
| Go Back to source page | Continue to Ontology report |