Enables Rab GDP-dissociation inhibitor activity and small GTPase binding activity. Involved in vesicle-mediated transport. Is active in synapse. Orthologous to human GDI2 (GDP dissociation inhibitor 2); PARTICIPATES IN Rab family mediated signaling pathway; INTERACTS WITH 2,3,7,8-tetrachlorodibenzodioxine; 2-ethoxyethanol; 2-methoxyethanol.
[Acrolein co-treated with methacrylaldehyde co-treated with alpha-pinene co-treated with Ozone] results in increased oxidation of GDI2 mRNA, [Air Pollutants results in increased abundance of [Acrolein co-treated with methacrylaldehyde co-treated with alpha-pinene co-treated with Ozone]] which results in increased oxidation of GDI2 mRNA
[Acrolein co-treated with methacrylaldehyde co-treated with alpha-pinene co-treated with Ozone] results in increased oxidation of GDI2 mRNA, [Air Pollutants results in increased abundance of [Acrolein co-treated with methacrylaldehyde co-treated with alpha-pinene co-treated with Ozone]] which results in increased oxidation of GDI2 mRNA
[bisphenol A co-treated with Fulvestrant] results in decreased methylation of GDI2 gene, [Phenolsulfonphthalein deficiency co-treated with bisphenol A deficiency] results in increased expression of GDI2 protein
[Methionine deficiency co-treated with Choline deficiency] inhibits the reaction [Folic Acid deficiency results in increased expression of GDI2 protein]
[Methionine deficiency co-treated with Choline deficiency] inhibits the reaction [Folic Acid deficiency results in increased expression of GDI2 protein]
[Methionine deficiency co-treated with Choline deficiency] inhibits the reaction [Folic Acid deficiency results in increased expression of GDI2 protein]
Reduction of nucleoside diphosphate kinase B, Rab GDP-dissociation inhibitor beta and histidine triad nucleotide-binding protein in fetal Down syndrome brain.