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Gene: Akr1c13 (aldo-keto reductase family 1, member C13) Mus musculus
Symbol: Akr1c13
Name: aldo-keto reductase family 1, member C13
Description: Predicted to have bile acid binding activity; ketosteroid monooxygenase activity; and oxidoreductase activity, acting on the CH-OH group of donors, NAD or NADP as acceptor. Predicted to be involved in glycoside metabolic process; progesterone metabolic process; and prostaglandin metabolic process. Predicted to localize to cytosol. Human ortholog(s) of this gene implicated in 46,XY sex reversal 8; B-lymphoblastic leukemia/lymphoma; T-cell acute lymphoblastic leukemia; and leukemia. Is expressed in gut and liver lobe. Orthologous to several human genes including AKR1C1 (aldo-keto reductase family 1 member C1); AKR1C2 (aldo-keto reductase family 1 member C2); and AKR1C3 (aldo-keto reductase family 1 member C3); INTERACTS WITH 1,2-dichloroethane; 2,3,7,8-tetrachlorodibenzodioxine; 4-hydroxyphenyl retinamide.
Type: protein-coding
RefSeq Status: VALIDATED
Also known as: aldo-keto reductase AKR1C13; aldo-keto reductase family 1 member C13; KIAA4014; mKIAA4014
No known orthologs. homologs ...
Related Pseudogenes: Gm18857  
Latest Assembly: GRCm38 - Mouse Genome Assembly GRCm38
Mouse AssemblyChrPosition (strand)SourceGenome Browsers
GRCm38 Ensembl134,191,150 - 4,205,596 (+)EnsemblGRCm38mm10GRCm38
GRCm38134,191,168 - 4,205,604 (+)NCBIGRCm38GRCm38mm10GRCm38
MGSCv37134,190,433 - 4,204,849 (+)NCBIGRCm37mm9NCBIm37
MGSCv36134,190,467 - 4,204,848 (+)NCBImm8
Celera134,167,107 - 4,181,516 (+)NCBICelera
Cytogenetic Map13A1NCBI
JBrowse: View Region in Genome Browser (JBrowse)

References - curated
References - uncurated


Position Markers
QTLs in Region (GRCm38)
miRNA Target Status



Nucleotide Sequences
Protein Sequences

Additional Information

External Database Links
More on Akr1c13
Alliance Gene
Ensembl Gene
JBrowse: mm9 mm10
MGI Report
NCBI Genome Data Viewer

RGD Object Information
RGD ID: 1624047
Created: 2007-05-02
Species: Mus musculus
Last Modified: 2020-03-25
Status: ACTIVE


RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.