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Gene: Aggf1 (angiogenic factor with G patch and FHA domains 1) Rattus norvegicus
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Symbol: Aggf1
Name: angiogenic factor with G patch and FHA domains 1
Description: Predicted to have nucleic acid binding activity. Predicted to be involved in cell adhesion; positive regulation of angiogenesis; and positive regulation of endothelial cell proliferation. Predicted to localize to extracellular region and perinuclear region of cytoplasm. Orthologous to human AGGF1 (angiogenic factor with G-patch and FHA domains 1); INTERACTS WITH 2,2',5,5'-tetrachlorobiphenyl; 2,4-dinitrotoluene; 2,6-dinitrotoluene.
Type: protein-coding
RefSeq Status: MODEL
Also known as: LOC310005; RGD1310888; similar to RIKEN cDNA 2010009L17
Orthologs:
Latest Assembly: Rnor_6.0 - RGSC Genome Assembly v6.0
Position:
Rat AssemblyChrPosition (strand)SourceGenome Browsers
JBrowseNCBIUCSCEnsembl
Rnor_6.0225,068,804 - 25,095,429 (-)NCBIRnor6.0Rnor_6.0rn6Rnor6.0
Rnor_6.0 Ensembl225,069,620 - 25,095,106 (-)EnsemblRnor6.0rn6Rnor6.0
Rnor_5.0244,227,286 - 44,253,899 (-)NCBIRnor5.0Rnor_5.0rn5Rnor5.0
RGSC_v3.4225,723,668 - 25,749,359 (-)NCBIRGSC3.4rn4RGSC3.4
RGSC_v3.1225,644,037 - 25,679,865 (-)NCBI
Celera222,684,509 - 22,711,225 (-)NCBICelera
Cytogenetic Map2q12NCBI
JBrowse: View Region in Genome Browser (JBrowse)
Model


Disease Annotations
Gene-Chemical Interaction Annotations
Gene Ontology Annotations
References - curated
References - uncurated

Genomics

Comparative Map Data
Position Markers
QTLs in Region (Rnor_6.0)
miRNA Target Status

Expression

RNA-SEQ Expression

Sequence

Nucleotide Sequences
Protein Sequences
Transcriptome

Strain Variation

Strain Sequence Variants (Rnor 6.0)
Damaging Variants

Additional Information

External Database Links
Nomenclature History
 
More on Aggf1
Alliance Gene
NCBI Gene
Ensembl Gene
JBrowse: rn5 rn6
NCBI Genome Data Viewer

RGD Object Information
RGD ID: 1310888
Created: 2005-01-12
Species: Rattus norvegicus
Last Modified: 2019-11-27
Status: ACTIVE



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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.