GENE - CHEMICAL INTERACTIONS REPORT
Note: Use of the qualifier "multiple interactions" designates that the annotated interaction
is comprised of a complex set of reactions and/or regulatory events, possibly involving
additional chemicals and/or gene products.
Object Symbol | Qualifier | Evidence | With | Reference | Source | Notes | Original Reference(s) | H2ax | multiple interactions | EXP | | 6480464 | CTD | Acetylcysteine inhibits the reaction [[Cadmium Chloride results in increased abundance of Cadmium] which results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [aristolochic acid I results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [tributyltin results in increased expression of H2AX protein modified form]; Acetylcysteine inhibits the reaction [yttrium chloride results in increased phosphorylation of H2AX protein] | PMID:26079211 PMID:33031904 PMID:35088107 PMID:36863539 | H2ax | increases expression | ISO | H2AX (Homo sapiens) | 6480464 | CTD | Acetylcysteine results in increased expression of H2AX protein | PMID:31715134 PMID:35123993 | H2ax | multiple interactions | ISO | H2AX (Homo sapiens) | 6480464 | CTD | Acetylcysteine inhibits the reaction [2,3,5-(triglutathion-S-yl)hydroquinone results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [2-hydroxy-3',5,5'-trimethoxychalcone results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [4-biphenylamine results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [4-methoxy-2,2,6,6-tetramethylpiperidinyl-1-oxy results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [5-(3,4,5-trimethoxybenzoyl)-4-methyl-2-(p-tolyl)imidazole results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [[Bortezomib co-treated with romidepsin] results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [[fludarabine co-treated with LAQ824] results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [Acetaminophen results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [alpha-terthienylmethanol results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [bisphenol A results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [captax results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [chloropicrin results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [cobaltous chloride promotes the reaction [nickel chloride results in increased phosphorylation of H2AX protein]]; Acetylcysteine inhibits the reaction [cobaltous chloride results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [Crizotinib results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [dorsomorphin results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [Doxorubicin results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [eprenetapopt results in increased expression of H2AX protein modified form]; Acetylcysteine inhibits the reaction [gingerenone A results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [Hydrogen Peroxide results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [Hydrogen Peroxide results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [hydroquinone results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [LAQ824 results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [manumycin results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [nickel chloride promotes the reaction [cobaltous chloride results in increased phosphorylation of H2AX protein]]; Acetylcysteine inhibits the reaction [nickel chloride results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [PCB 180 metabolite results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [phenanthridone results in increased expression of H2AX protein modified form]; Acetylcysteine inhibits the reaction [Plant Extracts results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [polyhexamethyleneguanidine results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [propylene dichloride results in increased phosphorylation of H2AX protein]; Acetylcysteine inhibits the reaction [scutebarbatine A results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [tomentosin results in increased expression of H2AX protein]; Acetylcysteine inhibits the reaction [usnic acid results in increased expression of H2AX protein] | PMID:15173094 PMID:15489221 PMID:18852132 PMID:19409983 PMID:21135414 PMID:22172632 PMID:23274516 PMID:26148434 PMID:28300663 PMID:28506552 PMID:28993908 PMID:29128613 PMID:29416738 PMID:30408460 PMID:30917517 PMID:31157515 PMID:31168028 PMID:31639374 PMID:31682804 PMID:31715134 PMID:31884112 PMID:32433928 PMID:33971260 PMID:35123993 PMID:35279910 PMID:35326237 PMID:37072049 | H2ax | multiple interactions | ISO | H2ax (Mus musculus) | 6480464 | CTD | Acetylcysteine inhibits the reaction [2,4-dibromophenol metabolite results in increased expression of H2AX protein modified form]; Acetylcysteine inhibits the reaction [diepoxybutane results in increased expression of H2AX protein modified form]; Acetylcysteine inhibits the reaction [quinone analog results in increased expression of H2AX protein modified form]; Acetylcysteine inhibits the reaction [Zinc Oxide analog results in increased activity of H2AX protein] | PMID:24614525 PMID:25633853 PMID:33961949 | | ||||||||||||||||||||||
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