GENE - CHEMICAL INTERACTIONS REPORT
Note: Use of the qualifier "multiple interactions" designates that the annotated interaction
is comprised of a complex set of reactions and/or regulatory events, possibly involving
additional chemicals and/or gene products.
Object Symbol | Qualifier | Evidence | With | Reference | Source | Notes | Original Reference(s) | Tnfsf11 | increases expression | ISO | Tnfsf11 (Mus musculus) | 6480464 | CTD | resveratrol results in increased expression of TNFSF11 mRNA | PMID:16813520 | Tnfsf11 | multiple interactions | ISO | TNFSF11 (Homo sapiens) | 6480464 | CTD | [resveratrol co-treated with CSF1 protein co-treated with TNFSF11 protein] results in increased expression of CD14 mRNA; [resveratrol co-treated with CSF1 protein co-treated with TNFSF11 protein] results in increased expression of CSF1R mRNA; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in decreased expression of ITGAL mRNA]; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in increased activity of ACP5 protein]; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in increased expression of CTSK mRNA]; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in increased expression of NFATC1 mRNA]; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in increased expression of TNFRSF11A mRNA]; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in increased expression of TNFRSF11A protein]; resveratrol inhibits the reaction [[CSF1 protein co-treated with TNFSF11 protein] results in increased localization of RELA protein] | PMID:16267019 | Tnfsf11 | multiple interactions | ISO | Tnfsf11 (Mus musculus) | 6480464 | CTD | resveratrol inhibits the reaction [norisoboldine inhibits the reaction [TNFSF11 protein results in increased expression of VEGFA mRNA]] | PMID:26221077 | | ||||||||||||||||||||||
| Go Back to source page | Continue to Ontology report |