Send us a Message



Submit Data |  Help |  Video Tutorials |  News |  Publications |  Download |  REST API |  Citing RGD |  Contact   

GENE - CHEMICAL INTERACTIONS REPORT

RGD ID: 3705
Species: Rattus norvegicus
RGD Object: Gene
Symbol: Slc2a2
Name: solute carrier family 2 member 2
Acc ID: CHEBI:27881
Term: resveratrol
Definition: A stilbenol that is stilbene in which the phenyl groups are substituted at positions 3, 5, and 4' by hydroxy groups.
Chemical ID: MESH:C059514
Note: Use of the qualifier "multiple interactions" designates that the annotated interaction is comprised of a complex set of reactions and/or regulatory events, possibly involving additional chemicals and/or gene products.
Object SymbolQualifierEvidenceWithReferenceSourceNotesOriginal Reference(s)
Slc2a2multiple interactionsEXP 6480464CTDResveratrol inhibits the reaction [bisphenol A results in decreased expression of SLC2A2 mRNA]; Resveratrol inhibits the reaction [Oxygen deficiency results in increased expression of SLC2A2 protein]PMID:25352008 PMID:37570835
Slc2a2increases expressionISOSLC2A2 (Homo sapiens)6480464CTDresveratrol results in increased expression of SLC2A2 mRNAPMID:21163946
Slc2a2multiple interactionsISOSlc2a2 (Mus musculus)6480464CTDResveratrol inhibits the reaction [Sodium Glutamate results in increased expression of SLC2A2 mRNA]; Resveratrol inhibits the reaction [Sodium Glutamate results in increased expression of SLC2A2 protein]PMID:28708105
Slc2a2multiple interactionsISOSLC2A2 (Homo sapiens)6480464CTD6-chloro-2,3,4,9-tetrahydro-1H-carbazole-1-carboxamide inhibits the reaction [SIRT1 protein promotes the reaction [resveratrol results in increased expression of SLC2A2 mRNA]]; NFE2L2 protein affects the reaction [resveratrol inhibits the reaction [Pyruvaldehyde affects the localization of SLC2A2 protein]]; resveratrol inhibits the reaction [Pyruvaldehyde affects the localization of SLC2A2 protein]; SIRT1 protein promotes the reaction [resveratrol results in increased expression of SLC2A2 mRNA]PMID:21163946 PMID:22917016
Go Back to source page   Continue to Ontology report