GENE - CHEMICAL INTERACTIONS REPORT
Note: Use of the qualifier "multiple interactions" designates that the annotated interaction
is comprised of a complex set of reactions and/or regulatory events, possibly involving
additional chemicals and/or gene products.
Object Symbol | Qualifier | Evidence | With | Reference | Source | Notes | Original Reference(s) | Dnm1l | multiple interactions | EXP | | 6480464 | CTD | [Blood Glucose deficiency co-treated with Oxygen deficiency] results in increased expression of DNM1L protein; genipin inhibits the reaction [[Blood Glucose deficiency co-treated with Oxygen deficiency] results in increased expression of DNM1L protein] | PMID:28477916 | Dnm1l | increases expression | ISO | Dnm1l (Rattus norvegicus) | 6480464 | CTD | Oxygen deficiency results in increased expression of DNM1L protein | PMID:25911325 PMID:27684054 | Dnm1l | multiple interactions | ISO | Dnm1l (Rattus norvegicus) | 6480464 | CTD | [[Oxygen deficiency co-treated with Blood Glucose deficiency] results in increased susceptibility to 6-(5-((cyclopropylamino)carbonyl)-3-fluoro-2-methylphenyl)-N-(2,2-dimethylprpyl)-3-pyridinecarboxamide] which results in decreased expression of DNM1L protein; [[Oxygen deficiency co-treated with Blood Glucose deficiency] results in increased susceptibility to SB 239063] which results in decreased expression of DNM1L protein; [Oxygen deficiency co-treated with Oxygen] results in increased expression of DNM1L protein; dorsomorphin inhibits the reaction [sappanone A inhibits the reaction [[Oxygen deficiency co-treated with Oxygen] results in increased expression of DNM1L protein]]; Ru 360 inhibits the reaction [Oxygen deficiency results in increased expression of DNM1L protein]; sappanone A inhibits the reaction [[Oxygen deficiency co-treated with Oxygen] results in increased expression of DNM1L protein]; Sevoflurane inhibits the reaction [Oxygen deficiency results in increased expression of DNM1L protein] | PMID:25911325 PMID:26116440 PMID:27684054 PMID:34358556 | | ||||||||||||||||||||||
| Go Back to source page | Continue to Ontology report |