Send us a Message



Submit Data |  Help |  Video Tutorials |  News |  Publications |  Download |  REST API |  Citing RGD |  Contact   

GENE - CHEMICAL INTERACTIONS REPORT

RGD ID: 732770
Species: Homo sapiens
RGD Object: Gene
Symbol: ABCG8
Name: ATP binding cassette subfamily G member 8
Acc ID: CHEBI:28119
Term: 2,3,7,8-tetrachlorodibenzodioxine
Definition: A polychlorinated dibenzodioxine that has formula C12H4Cl4O2.
Chemical ID: MESH:D013749
Note: Use of the qualifier "multiple interactions" designates that the annotated interaction is comprised of a complex set of reactions and/or regulatory events, possibly involving additional chemicals and/or gene products.
Object SymbolQualifierEvidenceWithReferenceSourceNotesOriginal Reference(s)
ABCG8decreases expressionEXP 6480464CTDTetrachlorodibenzodioxin results in decreased expression of ABCG8 mRNAPMID:20106945 PMID:26159488
ABCG8increases expressionEXP 6480464CTDTetrachlorodibenzodioxin results in increased expression of ABCG8 mRNAPMID:21632981
ABCG8multiple interactionsEXP 6480464CTD[Endosulfan co-treated with Tetrachlorodibenzodioxin] results in decreased expression of ABCG8 mRNAPMID:26159488
ABCG8decreases expressionISOAbcg8 (Rattus norvegicus)6480464CTDTetrachlorodibenzodioxin results in decreased expression of ABCG8 mRNAPMID:21215274
ABCG8decreases expressionISOAbcg8 (Mus musculus)6480464CTDTetrachlorodibenzodioxin results in decreased expression of ABCG8 mRNAPMID:28213091
ABCG8increases expressionISOAbcg8 (Mus musculus)6480464CTDTetrachlorodibenzodioxin results in increased expression of ABCG8 mRNAPMID:25975270
ABCG8multiple interactionsISOAbcg8 (Mus musculus)6480464CTD[NFE2L2 gene mutant form results in increased susceptibility to Tetrachlorodibenzodioxin] which results in decreased expression of ABCG8 mRNA; [Tetrachlorodibenzodioxin co-treated with bisphenol A co-treated with 2,4,5,2',4',5'-hexachlorobiphenyl co-treated with Diethylhexyl Phthalate] promotes the reaction [Dietary Fats results in increased expression of ABCG8 mRNA]PMID:21846477 PMID:23756648
Go Back to source page   Continue to Ontology report