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ONTOLOGY REPORT - ANNOTATIONS


Term:hyperandrogenism
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Accession:DOID:11613 term browser browse the term
Definition:A condition caused by the excessive secretion of ANDROGENS from the ADRENAL CORTEX; the OVARIES; or the TESTES. The clinical significance in males is negligible. In women, the common manifestations are HIRSUTISM and VIRILISM as seen in patients with POLYCYSTIC OVARY SYNDROME and ADRENOCORTICAL HYPERFUNCTION.
Synonyms:exact_synonym: hyperandrogenization syndrome
 primary_id: MESH:D017588
 alt_id: RDO:0003503
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hyperandrogenism term browser
Symbol Object Name JBrowse Chr Start Stop Reference
G Bmpr2 bone morphogenetic protein receptor type 2 JBrowse link 9 66,568,074 66,683,019 RGD:11554173
G Il6st interleukin 6 signal transducer JBrowse link 2 44,279,199 44,319,427 RGD:1625428
G Pparg peroxisome proliferator-activated receptor gamma JBrowse link 4 147,274,055 147,399,383 RGD:8553031
Mullerian aplasia and hyperandrogenism term browser
Symbol Object Name JBrowse Chr Start Stop Reference
G Wnt4 Wnt family member 4 JBrowse link 5 155,649,238 155,668,065 RGD:7240710
RGD:8554872

Term paths to the root
Path 1
Term Annotations click to browse term
  disease 15503
    syndrome 5771
      Adrenogenital Syndrome 25
        hyperandrogenism 4
          HAIR-AN syndrome 0
          Hyperandrogenism, Nonclassic Type, due to 21-Hydroxylase Deficiency 0
          Mullerian aplasia and hyperandrogenism 1
Path 2
Term Annotations click to browse term
  disease 15503
    Developmental Diseases 8880
      Congenital, Hereditary, and Neonatal Diseases and Abnormalities 7677
        Congenital Abnormalities 3811
          Urogenital Abnormalities 237
            sex development disorder 103
              Adrenogenital Syndrome 25
                hyperandrogenism 4
                  HAIR-AN syndrome 0
                  Hyperandrogenism, Nonclassic Type, due to 21-Hydroxylase Deficiency 0
                  Mullerian aplasia and hyperandrogenism 1
paths to the root

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RGD is funded by grant HL64541 from the National Heart, Lung, and Blood Institute on behalf of the NIH.