The Chemical Entities of Biological Interest (ChEBI) ontology is downloaded weekly from EMBL-EBI at http://www.ebi.ac.uk/chebi/. The data is made available under the Creative Commons License (CC BY 3.0, http://creativecommons.org/licenses/by/3.0/). For more information see: Degtyarenko et al. (2008) ChEBI: a database and ontology for chemical entities of biological interest. Nucleic Acids Res. 36, D344–D350.
Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of BECN1 protein]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of BECN1 mRNA]]
[Octreotide results in increased activity of SSTR2 protein] which results in increased activity of CASP3 protein; Octreotide results in increased activity of CASP3 protein Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in increased cleavage of CASP3 protein]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in increased cleavage of CASP3 protein]]
Octreotide inhibits the reaction [FSHB protein results in increased expression of CYP11A1 mRNA] NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of CYP11A1 mRNA]]
NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased abundance of Cyclic AMP]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased abundance of Progesterone]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of CYP11A1 mRNA]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of HSD3B2 mRNA]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of STAR mRNA]] Octreotide inhibits the reaction [FSHB protein results in increased abundance of Cyclic AMP]; Octreotide inhibits the reaction [FSHB protein results in increased abundance of Estradiol]; Octreotide inhibits the reaction [FSHB protein results in increased abundance of Progesterone]; Octreotide inhibits the reaction [FSHB protein results in increased expression of CYP11A1 mRNA]; Octreotide inhibits the reaction [FSHB protein results in increased expression of HSD3B2 mRNA]; Octreotide inhibits the reaction [FSHB protein results in increased expression of STAR mRNA]
Octreotide results in decreased secretion of GH1 protein Octreotide results in decreased expression of GH protein; Octreotide results in decreased expression of GH1 protein Octreotide affects the expression of GH1 protein [Octreotide co-treated with Sodium Glutamate] results in decreased expression of and results in decreased secretion of GH1 protein
Octreotide affects the reaction [SSTR1 protein affects the reaction [[Nitrogen co-treated with Iodoacetic Acid] results in increased expression of GRK1 mRNA]]; Octreotide inhibits the reaction [[Nitrogen co-treated with Iodoacetic Acid] results in increased expression of GRK1 mRNA]
Octreotide promotes the reaction [BMP2 protein results in increased expression of ID1 mRNA]; Octreotide promotes the reaction [BMP4 protein results in increased expression of ID1 mRNA]; Octreotide promotes the reaction [BMP6 protein results in increased expression of ID1 mRNA]; Octreotide promotes the reaction [BMP7 protein results in increased expression of ID1 mRNA]
Octreotide affects the secretion of INS protein Octreotide results in decreased expression of INS protein Octreotide results in decreased secretion of INS protein Octreotide inhibits the reaction [Hydrocortisone results in increased expression of INS protein]
Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of MAP1LC3B protein]; SIRT1 promotes the reaction [SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of MAP1LC3B protein]]]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of MAP1LC3B protein]]; YAP1 promotes the reaction [SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of MAP1LC3B protein]]]
NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased abundance of Cyclic AMP]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased abundance of Progesterone]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of CYP11A1 mRNA]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of HSD3B2 mRNA]]; NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of STAR mRNA]]
Octreotide results in decreased expression of PRL protein Colforsin inhibits the reaction [Octreotide results in decreased secretion of PRL protein]; Octreotide inhibits the reaction [Colforsin results in increased expression of PRL protein]; Pertussis Toxin inhibits the reaction [Octreotide results in decreased secretion of PRL protein] Octreotide affects the expression of PRL protein
Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of SIRT1 mRNA]; SIRT1 promotes the reaction [SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of MAP1LC3B protein]]]; SIRT1 promotes the reaction [SNHG12 mRNA results in increased susceptibility to Octreotide]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of SIRT1 mRNA]]
Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of SQSTM1 protein]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of SQSTM1 mRNA]]
Octreotide affects the reaction [SSTR1 protein affects the reaction [[Nitrogen co-treated with Iodoacetic Acid] results in increased expression of GRK1 mRNA]]
[Octreotide results in increased activity of SSTR2 protein] which results in increased activity of CASP3 protein SSTR2 protein affects the susceptibility to Octreotide [Octreotide results in increased activity of SSTR2 protein] which results in increased expression of KRT18 protein modified form
Octreotide inhibits the reaction [FSHB protein results in increased expression of STAR mRNA] NOG protein inhibits the reaction [Octreotide inhibits the reaction [FSHB protein results in increased expression of STAR mRNA]]
Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of TAF15 mRNA]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of TAF15 mRNA]]
Octreotide results in decreased expression of TNF mRNA Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in increased secretion of TNF protein]
Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of YAP1 mRNA]; SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of YAP1 mRNA]]; YAP1 promotes the reaction [SNHG12 mRNA promotes the reaction [Octreotide inhibits the reaction [[[Oxygen deficiency co-treated with Blood Glucose deficiency] co-treated with [Oxygen co-treated with Blood Glucose]] results in decreased expression of MAP1LC3B protein]]]; YAP1 promotes the reaction [SNHG12 mRNA results in increased susceptibility to Octreotide]
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