The Chemical Entities of Biological Interest (ChEBI) ontology is downloaded weekly from EMBL-EBI at http://www.ebi.ac.uk/chebi/. The data is made available under the Creative Commons License (CC BY 3.0, http://creativecommons.org/licenses/by/3.0/). For more information see: Degtyarenko et al. (2008) ChEBI: a database and ontology for chemical entities of biological interest. Nucleic Acids Res. 36, D344–D350.
ABCA1 protein promotes the reaction [anacetrapib results in increased export of Cholesterol, HDL]; ABCA1 protein promotes the reaction [Niacin results in increased export of Cholesterol, HDL] ABCA1 gene polymorphism affects the abundance of Cholesterol, HDL
ABCG1 protein promotes the reaction [anacetrapib results in increased export of Cholesterol, HDL]; ABCG1 protein promotes the reaction [Niacin results in increased export of Cholesterol, HDL]
2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one inhibits the reaction [[Cholesterol, HDL results in increased phosphorylation of and results in increased activity of AKT1 protein] which results in decreased degradation of NOS3 protein]; 2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one inhibits the reaction [Cholesterol, HDL results in increased phosphorylation of and results in increased activity of AKT1 protein]; [Cholesterol, HDL results in increased phosphorylation of and results in increased activity of AKT1 protein] which results in decreased degradation of NOS3 protein; Cholesterol, HDL results in increased phosphorylation of and results in increased activity of AKT1 protein
PON1 protein binds to [APOA1 protein binds to Cholesterol, HDL] Fatty Acids, Unsaturated affects the reaction [APOA1 promoter SNP affects the abundance of Cholesterol, HDL] APOA1 protein inhibits the reaction [Lipopolysaccharides results in decreased abundance of Cholesterol, HDL] APOA1 protein results in increased abundance of Cholesterol, HDL
APOE gene polymorphism affects the abundance of Cholesterol, HDL [[Air Pollutants results in increased abundance of Particulate Matter] promotes the reaction [APOE protein affects the susceptibility to Dietary Fats]] which affects the abundance of Cholesterol, HDL; [APOE gene mutant form affects the susceptibility to Folic Acid] which results in increased abundance of Cholesterol, HDL analog; [APOE gene mutant form results in increased susceptibility to Dietary Fats] which results in decreased abundance of Cholesterol, HDL; N-butyloxycarbonyl-methionyl-aspartyl-glycyl-cysteinyl-glutamyl-leucine inhibits the reaction [[[Air Pollutants results in increased abundance of Particulate Matter] promotes the reaction [APOE protein affects the susceptibility to Dietary Fats]] which affects the abundance of Cholesterol, HDL] APOE gene polymorphism results in increased abundance of Cholesterol, HDL
[Niacin results in decreased expression of and results in decreased activity of CERT1 protein] which results in increased abundance of Cholesterol, HDL
Dietary Fats affects the reaction [LIPC promoter SNP affects the abundance of Cholesterol, HDL] LIPC gene polymorphism affects the abundance of Cholesterol, HDL; LIPC promoter SNP affects the abundance of Cholesterol, HDL
2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one inhibits the reaction [Cholesterol, HDL results in increased phosphorylation of and results in increased activity of MAPK1 protein]; Cholesterol, HDL results in increased phosphorylation of and results in increased activity of MAPK1 protein
2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one inhibits the reaction [Cholesterol, HDL results in increased phosphorylation of and results in increased activity of MAPK3 protein]; Cholesterol, HDL results in increased phosphorylation of and results in increased activity of MAPK3 protein
[Arsenic results in increased expression of and results in increased secretion of MMP9 protein] which results in decreased abundance of Cholesterol, HDL
2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one inhibits the reaction [Cholesterol, HDL results in decreased degradation of NOS3 protein]; 2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one inhibits the reaction [[Cholesterol, HDL results in increased phosphorylation of and results in increased activity of AKT1 protein] which results in decreased degradation of NOS3 protein]; [Cholesterol, HDL results in increased phosphorylation of and results in increased activity of AKT1 protein] which results in decreased degradation of NOS3 protein Cholesterol, HDL results in increased expression of NOS3 protein
Cholesterol, HDL results in increased activity of PON1 protein PON1 gene polymorphism affects the reaction [Atorvastatin results in increased abundance of Cholesterol, HDL]; PON1 gene polymorphism affects the reaction [Simvastatin results in increased abundance of Cholesterol, HDL]; PON1 gene SNP affects the reaction [Atorvastatin results in increased abundance of Cholesterol, HDL]; PON1 gene SNP affects the reaction [Simvastatin results in increased abundance of Cholesterol, HDL]; PON1 protein affects the reaction [Atorvastatin results in increased abundance of Cholesterol, HDL]; PON1 protein affects the reaction [Simvastatin results in increased abundance of Cholesterol, HDL] PON1 gene polymorphism affects the abundance of Cholesterol, HDL; PON1 polymorphism affects the abundance of Cholesterol, HDL PON1 protein binds to [APOA1 protein binds to Cholesterol, HDL]
Niacin promotes the reaction [SCARB1 protein results in increased export of Cholesterol, HDL] Fenofibrate inhibits the reaction [SCARB1 protein results in increased export of Cholesterol, HDL]; SDZ HDL 376 analog inhibits the reaction [SCARB1 protein results in increased export of Cholesterol, HDL]; SDZ HDL 376 inhibits the reaction [SCARB1 protein results in increased export of Cholesterol, HDL]
TNF gene polymorphism affects the abundance of Cholesterol, HDL Cholesterol, HDL inhibits the reaction [[lipopolysaccharide, E coli O55-B5 co-treated with Carbon Tetrachloride] results in increased expression of TNF protein]
High-density lipoprotein inhibits the reaction [cholesterol increases expression of ITGAM protein on monocytes and granulocytes] High-density lipoprotein inhibits the reaction [PMA increases expression of ITGAM protein on monocytes]
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