The Chemical Entities of Biological Interest (ChEBI) ontology is downloaded weekly from EMBL-EBI at http://www.ebi.ac.uk/chebi/. The data is made available under the Creative Commons License (CC BY 3.0, http://creativecommons.org/licenses/by/3.0/). For more information see: Degtyarenko et al. (2008) ChEBI: a database and ontology for chemical entities of biological interest. Nucleic Acids Res. 36, D344–D350.
N(6)-(delta(2)-isopentenyl)adenine results in increased expression of S100P mRNA; N(6)-(delta(2)-isopentenyl)adenine results in increased expression of S100P protein
N(6)-(delta(2)-isopentenyl)adenine results in decreased expression of RGS16 mRNA N(6)-(delta(2)-isopentenyl)adenine results in increased expression of RGS16 mRNA
AHR mutant form inhibits the reaction [[Estradiol co-treated with triclocarban] results in increased expression of CYP1A1 mRNA] triclocarban results in increased expression of AHR mRNA; triclocarban results in increased expression of AHR protein
triclocarban analog promotes the reaction [Testosterone results in increased activity of AR protein]; triclocarban promotes the reaction [Dihydrotestosterone binds to and results in increased activity of AR protein]; triclocarban promotes the reaction [Testosterone results in increased activity of AR protein]; triclocarban promotes the reaction [Trenbolone Acetate results in increased activity of AR protein]
[bisphenol A co-treated with triclocarban] results in increased expression of CYP1A1 mRNA; [butylparaben co-treated with triclocarban] results in increased expression of CYP1A1 mRNA; [Estradiol co-treated with triclocarban] results in increased expression of CYP1A1 mRNA; [Genistein co-treated with triclocarban] results in increased expression of CYP1A1 mRNA; AHR mutant form inhibits the reaction [[Estradiol co-treated with triclocarban] results in increased expression of CYP1A1 mRNA]; ESR1 mutant form inhibits the reaction [[Estradiol co-treated with triclocarban] results in increased expression of CYP1A1 mRNA]; triclocarban inhibits the reaction [Tetrachlorodibenzodioxin results in increased activity of CYP1A1 protein]; triclocarban inhibits the reaction [Tetrachlorodibenzodioxin results in increased expression of CYP1A1 mRNA] triclocarban results in increased expression of CYP1A1 mRNA
ESR1 protein affects the reaction [triclocarban results in increased expression of CYP1B1 mRNA]; triclocarban promotes the reaction [bisphenol A results in increased expression of CYP1B1 mRNA]; triclocarban promotes the reaction [Genistein results in increased expression of CYP1B1 mRNA]
NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of CYP2B10 mRNA]; NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of CYP2B10 protein] triclocarban results in increased expression of CYP2B10 mRNA; triclocarban results in increased expression of CYP2B10 protein ESR1 protein affects the reaction [triclocarban results in increased expression of CYP2B6 mRNA]
ESR1 mutant form inhibits the reaction [[Estradiol co-treated with triclocarban] results in increased expression of CYP1A1 mRNA]; ESR1 protein affects the reaction [triclocarban results in increased expression of CYP1B1 mRNA]; ESR1 protein affects the reaction [triclocarban results in increased expression of CYP2B6 mRNA]; triclocarban analog promotes the reaction [Estradiol results in increased activity of ESR1 protein]; triclocarban promotes the reaction [Estradiol results in increased activity of ESR1 protein] triclocarban results in decreased expression of ESR1 mRNA; triclocarban results in decreased expression of ESR1 protein
triclocarban results in decreased expression of GJA1 protein modified form triclocarban results in decreased expression of and affects the localization of GJA1 protein
triclocarban inhibits the reaction [lipopolysaccharide, Escherichia coli O111 B4 results in increased secretion of IL6 protein] triclocarban results in increased expression of IL6 mRNA
NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of UGT1A1 mRNA]; NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of UGT1A3 mRNA]; NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of UGT1A4 mRNA]; NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of UGT1A6 mRNA]; NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of UGT1A9 mRNA] NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of CYP2B10 mRNA]; NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of CYP2B10 protein]
triclocarban results in decreased activity of SLC5A5 protein triclocarban inhibits the reaction [SLC5A5 protein results in increased uptake of Iodides]; triclocarban inhibits the reaction [SLC5A5 protein results in increased uptake of Iodine]
triclocarban results in increased expression of UGT1A1 mRNA NR1I3 gene mutant form inhibits the reaction [triclocarban results in increased expression of UGT1A1 mRNA]