The Chemical Entities of Biological Interest (ChEBI) ontology is downloaded weekly from EMBL-EBI at http://www.ebi.ac.uk/chebi/. The data is made available under the Creative Commons License (CC BY 3.0, http://creativecommons.org/licenses/by/3.0/). For more information see: Degtyarenko et al. (2008) ChEBI: a database and ontology for chemical entities of biological interest. Nucleic Acids Res. 36, D344–D350.
A member of the class of benzamides resulting from the formal condensation of 4-amino-5-chloro-2-methoxybenzoic acid with the primary amino group of N,N-diethylethane-1,2-diamine.
Metoclopramide binds to and results in decreased activity of ACHE protein; Metoclopramide inhibits the reaction [mipafox binds to and results in decreased activity of ACHE protein]; Metoclopramide inhibits the reaction [mipafox results in decreased activity of ACHE protein]; Metoclopramide inhibits the reaction [Paraoxon binds to and results in decreased activity of ACHE protein]; Metoclopramide inhibits the reaction [Paraoxon results in decreased activity of ACHE protein]
Metoclopramide affects the expression of AR protein [Metoclopramide results in decreased abundance of Testosterone] which results in decreased expression of AR protein Metoclopramide results in increased expression of AR protein
Metoclopramide inhibits the reaction [mipafox binds to and results in decreased activity of BCHE protein]; Metoclopramide inhibits the reaction [Paraoxon binds to and results in decreased activity of BCHE protein]
Metoclopramide inhibits the reaction [Cisplatin results in increased activity of CASP3 protein]; Metoclopramide inhibits the reaction [Etoposide results in increased activity of CASP3 protein]; Metoclopramide inhibits the reaction [Malathion results in increased activity of and results in increased cleavage of CASP3 protein]; Metoclopramide inhibits the reaction [Paclitaxel results in increased activity of CASP3 protein]; Metoclopramide inhibits the reaction [Paraoxon results in increased activity of and results in increased cleavage of CASP3 protein]; Metoclopramide inhibits the reaction [TNF protein results in increased activity of CASP3 protein]
[Metoclopramide binds to and results in decreased activity of CHRM3 protein] which results in decreased susceptibility to Carbachol; Metoclopramide inhibits the reaction [4-diphenylacetoxy-N-methylpiperidine methobromide binds to CHRM3 protein] Metoclopramide binds to CHRM3 protein
Metoclopramide binds to and results in decreased activity of DRD2 protein Metoclopramide binds to DRD2 protein Metoclopramide results in increased expression of DRD2 protein
[Dexamethasone co-treated with Metoclopramide] results in decreased activity of HTR3A protein; [Methylprednisolone co-treated with Metoclopramide] results in decreased activity of HTR3A protein; Metoclopramide binds to and results in decreased activity of HTR3A protein; Metoclopramide inhibits the reaction [GR 65630 binds to HTR3A protein] Metoclopramide results in decreased activity of HTR3A protein Metoclopramide binds to HTR3A protein
Metoclopramide inhibits the reaction [Malathion results in increased cleavage of PARP1 protein]; Metoclopramide inhibits the reaction [Paraoxon results in increased cleavage of PARP1 protein]
Lisuride inhibits the reaction [Metoclopramide results in increased secretion of PRL protein]; Metoclopramide results in increased expression of and results in increased secretion of PRL protein Metoclopramide affects the expression of PRL protein Metoclopramide results in increased expression of PRL protein